colloid ZINC
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| Zinc Colloid |
| Ultra Colloidal ZINC |
Why Zinc?
The 14th most Concentrated Human Body Element vital in all
healing processes:
Depleted by:
alcoholism, smoking, strenuous activity.
90% of the western world is deficient in Zinc. The 14th
element in relative concentration in the human body, though
required in high abundance is not sufficiently available EVEN
IN SUPPLEMENTAL FORMS.
Why? Because like other mostly non soluble metallic elements
they exist primarily in colloidal form (non-soluble particles in
liquid suspension} which in water supplies have a diminished
ability to carry concentrations with only a fraction of the
more soluble elements. The human body requirement for zinc
in this less available colloidal form is further diminished because
commercial water processes remove colloids, worse still the
farm soils are seriously deplete and the last resource are those
commercially compressed least bio-available tablet
supplements.
Not surprising that whole populations are starved for this
important nutrient. Even the less soluble trace elements like
copper the # 20 out of 60 known body elements is less likely
deplete because it is 32 times more trace than zinc. Calcium #5,
Magnesium #11 and Zinc #14 are the three foundational elements
of human health. Of the three, Zinc depletion is likely the one
causing the most trouble. Zinc is not a human dietary element
safely forgotten. The following research conclusions are good
examples why:
The 14th most Concentrated Human Body Element vital in all
healing processes:
- Vital in body production of insulin . (See research- Dr.
Cass Ingram) - Reduces prostate swelling.
- Used in body building diets.
- Used in vein and artery plaque removal.
- Relieves angina.
- Fights heart disease . (See below article)
- Boosts testosterone production.
- Enhances fertility.
Depleted by:
alcoholism, smoking, strenuous activity.
90% of the western world is deficient in Zinc. The 14th
element in relative concentration in the human body, though
required in high abundance is not sufficiently available EVEN
IN SUPPLEMENTAL FORMS.
Why? Because like other mostly non soluble metallic elements
they exist primarily in colloidal form (non-soluble particles in
liquid suspension} which in water supplies have a diminished
ability to carry concentrations with only a fraction of the
more soluble elements. The human body requirement for zinc
in this less available colloidal form is further diminished because
commercial water processes remove colloids, worse still the
farm soils are seriously deplete and the last resource are those
commercially compressed least bio-available tablet
supplements.
Not surprising that whole populations are starved for this
important nutrient. Even the less soluble trace elements like
copper the # 20 out of 60 known body elements is less likely
deplete because it is 32 times more trace than zinc. Calcium #5,
Magnesium #11 and Zinc #14 are the three foundational elements
of human health. Of the three, Zinc depletion is likely the one
causing the most trouble. Zinc is not a human dietary element
safely forgotten. The following research conclusions are good
examples why:
increase your risk of heart attack. By triggering inflammation and
lowering the body’s level of cardio-protective compounds, it can lead to
clogged arteries…or worse.
HIGH DOSE ZINC TO TERMINATE ANGINA PECTORIS: A REVIEW
AND HYPOTHESIS FOR ACTION BY ICAM INHIBITION
By George A. Eby, M.S. and William W. Halcomb, D.O.
E-Mail: george.eby@george-eby-research.com
Web Site: http://george-eby-research.com
Posted: 12 June 2005
Abstract
We reviewed the literature related to the effects of high dose zinc in
arteriosclerosis-induced angina pectoris. Lipid peroxidation and LDL
oxidation are believed to be critical for arteriosclerosis, and consequently
angina pectoris. Administration of biologically available zinc was a
beneficial treatment in a significant percentage of patients with severely
symptomatic, inoperable atherosclerotic disease. In these patients, there
was no difference in zinc concentration between patients with and without
atherosclerosis in whole blood, erythocytes or hair, but there was a major
difference between normal aorta and diseased aortas (40.6 ppm zinc in
normal aorta vs. 23.2 ppm zinc in atherosclerotic aorta, 40.6 ppm zinc in
normal aorta vs. 19.4 ppm zinc in atherosclerotic aneurysm aorta), and no
difference between normal and aneurysm aorta, although copper was low
in aneurysm aorta. Medication with high dose zinc sulfate to raise zinc
serum concentrations from 95 micrograms per deciliter to 177 micrograms
per deciliter resulted in objective improvement in 12 of 16 of these
patients, including a patient that also had Raynard’s disease. Long term
environmental exposure to zinc resulted in a 40% reduction in the
incidence of angina of effort compared to people not exposed to
environmental zinc (P<0.01) and a 40% reduction in the incidence of
probable ischemia in exercise (P<0.001). Lead had no effect while cadmium
exposure resulted in more than tripling the incidence of angina of effort
(P<0.001). The antioxidative action of zinc prevents oxidation of LDL
cholesterol and consequently stops the main mechanism of atherogenesis.
Zinc blocks calcium and its several actions on atherogenesis. Increased
amounts of cytotoxic cytokines such as TNF-alpha, IL-beta and IL-8,
often produced in the elderly, are blocked by high dose zinc. We
hypothesize that higher serum concentrations of LDL cholesterol resulting
from administration of 300 mg of zinc per day is caused by a release of
low density cholesterol from cardiovascular tissues, beneficially flushing it
into the serum where it is readily observed, thus decreasing
arteriosclerosis, increasing circulation, terminating angina pectoris and
restoring more youthful cardiac function. Although prevention of
cholesterol-induced arteriosclerosis by zinc is predicted from findings
related to oxidative stress and lipid peroxidation, removal of LDL might be
attributable to action of ionic zinc on ICAM inhibition. In stark contrast to
current practice, high dose zinc should be considered as basic in the
strategy of prophylaxis and therapy of the atherosclerosis process to
terminate angina pectoris and restore youthful cardiac function.
Methods and Procedures
In 1981 while conducting a clinical trial using 23 mg of zinc from zinc
gluconate each two hours to treat common colds,(10) a 65 year old man
with severe, disabling angina pectoris evaded our selection criteria and
received zinc. His cold did not respond to zinc and he consumed 300 mg of
zinc on the first day and 276 mg of zinc on the following days of the 7 day
trial. No copper supplements were given to alter the effects of high dose
zinc.
Results
The patient returned a few days after the trial was over and wanted to
know whether he had received zinc or placebo. After 5 days on zinc, his
angina pectoris pain, very poorly controlled with any medication including
nitroglycerin, vanished for the first time in 15 years. Indeed, his blood
pressure was normal and he showed no outward evidence of coronary
damage. After his zinc therapy, he went snow skiing in the mountains of
Colorado for the first time in over 15 years and had no cardiac discomfort.
He obtained a new job and felt fine working hard all day.
lowering the body’s level of cardio-protective compounds, it can lead to
clogged arteries…or worse.
HIGH DOSE ZINC TO TERMINATE ANGINA PECTORIS: A REVIEW
AND HYPOTHESIS FOR ACTION BY ICAM INHIBITION
By George A. Eby, M.S. and William W. Halcomb, D.O.
E-Mail: george.eby@george-eby-research.com
Web Site: http://george-eby-research.com
Posted: 12 June 2005
Abstract
We reviewed the literature related to the effects of high dose zinc in
arteriosclerosis-induced angina pectoris. Lipid peroxidation and LDL
oxidation are believed to be critical for arteriosclerosis, and consequently
angina pectoris. Administration of biologically available zinc was a
beneficial treatment in a significant percentage of patients with severely
symptomatic, inoperable atherosclerotic disease. In these patients, there
was no difference in zinc concentration between patients with and without
atherosclerosis in whole blood, erythocytes or hair, but there was a major
difference between normal aorta and diseased aortas (40.6 ppm zinc in
normal aorta vs. 23.2 ppm zinc in atherosclerotic aorta, 40.6 ppm zinc in
normal aorta vs. 19.4 ppm zinc in atherosclerotic aneurysm aorta), and no
difference between normal and aneurysm aorta, although copper was low
in aneurysm aorta. Medication with high dose zinc sulfate to raise zinc
serum concentrations from 95 micrograms per deciliter to 177 micrograms
per deciliter resulted in objective improvement in 12 of 16 of these
patients, including a patient that also had Raynard’s disease. Long term
environmental exposure to zinc resulted in a 40% reduction in the
incidence of angina of effort compared to people not exposed to
environmental zinc (P<0.01) and a 40% reduction in the incidence of
probable ischemia in exercise (P<0.001). Lead had no effect while cadmium
exposure resulted in more than tripling the incidence of angina of effort
(P<0.001). The antioxidative action of zinc prevents oxidation of LDL
cholesterol and consequently stops the main mechanism of atherogenesis.
Zinc blocks calcium and its several actions on atherogenesis. Increased
amounts of cytotoxic cytokines such as TNF-alpha, IL-beta and IL-8,
often produced in the elderly, are blocked by high dose zinc. We
hypothesize that higher serum concentrations of LDL cholesterol resulting
from administration of 300 mg of zinc per day is caused by a release of
low density cholesterol from cardiovascular tissues, beneficially flushing it
into the serum where it is readily observed, thus decreasing
arteriosclerosis, increasing circulation, terminating angina pectoris and
restoring more youthful cardiac function. Although prevention of
cholesterol-induced arteriosclerosis by zinc is predicted from findings
related to oxidative stress and lipid peroxidation, removal of LDL might be
attributable to action of ionic zinc on ICAM inhibition. In stark contrast to
current practice, high dose zinc should be considered as basic in the
strategy of prophylaxis and therapy of the atherosclerosis process to
terminate angina pectoris and restore youthful cardiac function.
Methods and Procedures
In 1981 while conducting a clinical trial using 23 mg of zinc from zinc
gluconate each two hours to treat common colds,(10) a 65 year old man
with severe, disabling angina pectoris evaded our selection criteria and
received zinc. His cold did not respond to zinc and he consumed 300 mg of
zinc on the first day and 276 mg of zinc on the following days of the 7 day
trial. No copper supplements were given to alter the effects of high dose
zinc.
Results
The patient returned a few days after the trial was over and wanted to
know whether he had received zinc or placebo. After 5 days on zinc, his
angina pectoris pain, very poorly controlled with any medication including
nitroglycerin, vanished for the first time in 15 years. Indeed, his blood
pressure was normal and he showed no outward evidence of coronary
damage. After his zinc therapy, he went snow skiing in the mountains of
Colorado for the first time in over 15 years and had no cardiac discomfort.
He obtained a new job and felt fine working hard all day.
Pages
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